Tissue factor (thromboplastin) release stimulates the extrinsic coagulation cascade, initially via the activation of factor VII. Noninfectious granulomas result from innate or adaptive immune responses to large or persistent antigens that can include foreign material (dusts), self (rheumatoid nodules), and unknown agents ( sarcoidosis). Preface The second edition of Robbins and Cotran Pathology Flash Cards is designed to reinforce the learning that begins with the study of other works in the Robbins family of textbooks, as well as in other clinical and basic science disciplines. School: University of Ibadan. The cut kidney surface shows a wedge-shaped pale area ( ▪) of infarction ( coagulative necrosis) with a red thromboembolus ( ▴) in a renal artery branch beneath the lesion. Download Robbins and Cotran Pathology Flash Cards E-Book PDF. What hormone delays this process?
Year Of exam: 1986. course code: ANAT201, ANAT203, ANAT205, ANAT204, ANAT206, ANAT202, ANAT301, ANAT302, ANAT303. The vascular occlusion likely resulted from the embolization of a fragment of the valvular vegetation. Robbins and Cotran Pathology Companion Workbook by Edward C. Klatt. What is the critical feature of pleuripotent stem cells? Questions A 20-year-old woman with systemic lupus erythematosus (SLE) develops chest wall and abdominal pain focally, with ischemia of the muscle and overlying skin. What are the risk factors for increased amounts of this pigment? Inflammation leads to vasodilation followed by increased vascular (mostly venular) permeability, with leakage of fluid into the interstitium. This style effectively doubles your learning opportunities.
When this occurs, one cell of every division retains the capacity for self-renewal; this is the functional definition of "stemness. " Reward Your Curiosity. Some mediators made during this process—represented as "A" in the figure—act through cell surface receptors with intrinsic kinase activity (i. e., with the ability to phosphorylate select protein substrates). Robbins and cotran pathology flash cards free download season 1. The appearance of tissue from a pericardial biopsy specimen is shown. Name this process, and give the mechanism by which the keratinocytes are eliminated (arrow). Damage to small vessels near the infarct caused bleeding into calyces that manifested as hematuria. Her coagulation times and platelet count are normal. Questions One week after an uncomplicated laparoscopic hernia repair, a 37-year-old pathologist elects to have the healing incision site biopsied for educational purposes.
Course code: PAT301, PAT401, PAT402, PAT402. He is known to be HIV-positive. Metalloproteinases (collagenases, gelatinases, stromelysins, and membrane-bound forms). Questions A 29-year-old man incurs a deep laceration to his lower chest and within minutes develops hypotension, tachycardia, dyspnea, and then cyanosis. Robbins and cotran pathology flash cards free download manager. At what point is cell cycle arrest most likely to occur in response to radiation? Department: For other's category. Author: Husain Sattar. His WBC count is 14, 720/mm (nl 4000/mm to 11, 000/mm), with a differential count of 76% neutrophils, 9% band forms, 11% lymphocytes, and 4% monocytes. Diminished blood supply leads to increased renin secretion by the hypoperfused kidney, which causes hypertension.
DOWNLOAD] ⚡️ (PDF) Robbins Basic Pathology: with STUDENT CONSULT Online Access (Robbins Pathology). 6 Diseases of the Immune System, Sheets. Interleukin-1, tumor necrosis factor, and interferon-γ promote this endothelial cell retraction. Use the cards to consolidate your learning and inspire further study. Each side includes a vignette with text, image, and questions.
Author: Dennis Kasper, Anthony Fauci, Stephen Hauser, Dan Longo, Larry Jameson, Joseph Loscalzo. Course code: CHEMICAL PATHOLOGY. What cellular change has occurred? There is extensive lipochrome (lipofuscin) pigment within the myocardial fibers. Her electrocardiogram shows reduced voltages.
The breast is a modified apocrine gland; lipid-rich secretions are formed through the budding of apical portions of epithelial cytoplasm. What is the likely cause of this finding? File size: 18788 KB. Extravasated fibrinogen polymerizes to form fibrin strands that produce the shaggy exudate. At left is Edward C. Klatt, MD, professor of Pathology, Department of Biomedical Sciences, Mercer University School of Medicine; the other guy is Rick Mitchell, MD, PhD, professor of Pathology and Health Sciences and Technology, Harvard Medical School. His brain MRI is shown. The major opsonins that bind to microbes and aid in their phagocytosis include IgG (phagocytes bind via Fc receptors) and the C3b complement fragment. Robbins and cotran pathology flash cards free download games. The epicardium shows a dull, shaggy fibrinous exudate as well as serous fluid pooling in the pericardial cavity. With all this tissue bleeding, do you expect the mean corpuscular volume (MCV) to decrease? Describe the findings. Collagen synthesis is driven by transforming growth factor-β. How might this form of cell death be involved in the cyclic sloughing of the endometrium (menses)? Questions A 58-year-old nonsmoker with normal lung function is killed in a hit-and-run accident. 200 67 122MB Read more.
SoundCloud wishes peace and safety for our community in Ukraine. Questions A 62-year-old woman with a 4-day history of productive cough and fever has a WBC count of 14, 260/mm (nl 3800/mm to 10, 800/mm), with 70% neutrophils and 10% band forms. Mitral stenosis can lead to left atrial enlargement with subsequent mural thrombosis followed by embolization with infarction. An exudate would have many cells and higher protein content. What underlying disease does she have? Questions A 71-year-old man has had chest pain for the past week. PLEASE NOTE: We do not host/store any copyrighted content on our website, it's a catalog of links that are already found on the internet. If the patient were to survive to the age of 20 years, what form of the disease would she have? Robbins Pathology Flash Cards | iPhone & iPad Game Reviews | AppSpy.com. What inflammatory cells are present in the center of the lesion? Although more common during the eighth and ninth decades, this man's disease became symptomatic when he was 44 years old. Relative amounts of pro-apoptotic and anti-apoptotic proteins regulate mitochondrial cytochrome c release, which controls the activation of intracellular caspases that degrade cellular elements via a proteolytic cascade. Insult: Adaptation, Injury, and Death, Sheets.
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